Numerical simulation of IL-8-based relative inflammation potentials of aerosol particles from vehicle exhaust and non-exhaust emission sources in Japan

Mizuo Kajino*, Satoko Kayaba*, Yasuhiro Ishihara*, Yoko Iwamoto, Tomoaki Okuda, Hiroshi Okochi

*この研究の対応する著者

研究成果: Article査読

抄録

Spatial distributions of interleukin-8 (IL-8)-based relative inflammation potentials (IP) of PM2.5 from vehicle exhaust and non-exhaust emission sources in Japan are derived using the meteorology–chemistry model (NHM-Chem) and laboratory experiments. In this study, IP is first defined as multiplying PM2.5 from different emission sectors by supernatant IL-8 concentrations released using PM2.5 samples, normalized to that of particle-free controls. The simulated IP of primary exhaust particles IP(E) accounts for 3%–30% of the total vehicle IP (exhaust + non-exhaust, primary + secondary), IP(V), which is low in densely populated regions (3%–15%) and high (5%–30%) in less populated regions, because there are fewer exhaust PM2.5 emitters (diesel trucks) in more populated regions. The contribution of IP(V) to IP of the total environmental PM2.5, IP(A), varied substantially in space by approximately 3–5 times (the contributions are greater in larger cities as there is more traffic). In our estimates, IP(V) is approximately one and two orders of magnitude higher than IP(E) and IP(T), the IP of fresh tire wear particles (TWPs), respectively. IP(T) has a minor contribution to IP(V) and IP(A). Recently, however, aged TWPs have been reported to be toxic; thus, the aging process of TWPs needs to be considered in the future.

本文言語English
論文番号100237
ジャーナルAtmospheric Environment: X
21
DOI
出版ステータスPublished - 2024 1月

ASJC Scopus subject areas

  • 環境科学一般
  • 大気科学

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